The Journal of Alternative and Complementary Medicine. Although estrogen replacement increases vaginal lubrication female sexual function restores vaginal epithelial integrity, this therapy may not be appropriate for all patients, due to associated risk of breast and endometrial cancer. These observations suggest that adrenergic nerves mediate the contractile response. Exogenously added norepinephrine caused a dose-dependent contraction in vaginal and clitoral tissues.
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This elegant study showed that pelvic nerve-stimulation caused an increase in vaginal blood flow, vaginal wall pressure, vaginal length, clitoral intracavernosal pressure and clitoral blood flow and a decrease in vaginal luminal pressure. The paucity of biological data may be attributed to lack of reliable experimental models and tools for the investigation of female sexual function, and to limited funding, which is critical for the female sexual function of experimental approaches. More importantly, female sexual dysfunction is associated with negative sexual relationship experiences. Hyperglycemia affects vaginal smooth muscle response to neurotransmission affecting multiple physiological pathways.
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The worries of pregnancy and demands of being a new mother may have similar effects. These observations suggest that NOS in vaginal tissue is regulated by androgens and estrogens in an opposite manner. Dramatic increase in capillary inflow in the submucosa overwhelms Na-reabsorption leading to ml of vaginal transudate, enhancing lubrication essential for pleasurable coitus. Untreated anxiety or depression can cause or contribute to sexual dysfunction, as can long-term stress and a history of sexual female sexual function. In summary, data derived from in vivo animal models indicate that vasoactive agents play a role in genital arousal.
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